Talk:Parent–offspring conflict

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Untitled[edit]

Submitted by copyright holder. Ian Pitchford 21:53, 14 Apr 2005 (UTC)

I removed the following paragraph from the article:

Within the offspring there will be sexual conflict between the genes from the father and those from the mother ^{[citation needed]}, with paternally derived genes activating to facilitate a greater demand for mother's milk during infancy ^{[citation needed]}. Evidence for this comes from Prader-Willi syndrome (a rare genetic defect in which infants only have a single paternal copy of chromosomal region 15q11-13) have a poor sucking response and weak cry. Conversely, infants with Angelman syndrome only have a single maternal copy of 15q11-13 and are active and display strong, but poorly coordinated, suckling (thus demonstrating that both versions of the gene are necessary for the infant). This latter effect is an instance of genomic imprinting in which the effects of genes differ depending on whether they are contributed by the father or the mother.

Two claims are tagged with Fact tags. The evidence presented here looks like original research. If anyone can find a source making these same claims, by all means restore the paragraph. 198.189.164.204 (talk) 01:39, 19 May 2009 (UTC)[reply]

A mistake in the article?[edit]

Regardng "This leads to the prediction that, other things being equal, POC will be stronger under half siblings (e.g., unrelated males father a female's successive offspring) than under full siblings."

That sounds more like O-O conflict to me.TylerTim (talk) 20:22, 8 December 2013 (UTC)[reply]

Hi TylerTim! I get what you mean, but if you check the second source, it clearly says: "An implication of POCT is that the presence of siblings will increase conflict in biological parent–child dyads, and that half siblings will increase that conflict more than full siblings." Lova Falk talk 08:23, 26 December 2013 (UTC)[reply]